A growing dysfunction in energy metabolism, such as might be caused by higher levels ofmitochondrial damage in cells, has been implicated in the progression of age-relatedneurodegenerative conditions. The neurons of the brain collectively require a lot of energy to operate, and thus it is reasonable to consider that disruptions in the processes that provide that energy are significant. Such disruptions are not the root causes of neurodegeneration, however, but rather secondary or later effects of an accumulation of the fundamental cell and tissue damage that causes aging. One possible compensatory approach to therapy, a treatment that doesn’t address the root causes but instead tries to modestly slow down their consequences, would involve…
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